Although the data on males is lacking (understandably so given the target market), there is some evidence that estrogen suppression with aromatase inhibitors causes the body to respond/adapt with an increase in sensitivity to estrogens, both by upregulating estrogen receptor levels and increasing E2 signaling via other pathways. This would not favor recovery. A lot of talk as of late on gyno caused AFTER PCT ended, when the PCT included an aromatase inhbitior. I am not knocking AR for coming up with this theory. Theories are good. But it is still a theory, and like all theories it will not be fact until it is proven and accepted. I am not so sure this one ultimately will be.
AI induced "estrogen hypersensitivty" aside, I am still inclined to think that AIs are an unnecessary addition given an environment that already favors lower E2 stimualtion and higher LH output.
Just my .02.
|