Quote:
Originally Posted by jinxie1  Jan, given that your balls are shut down, and your hCG is largely for cosmetic purposes, I dont think folks can infer much from your doses in terms of armotasization, or, for that matter, impact on T levels from hCG. |
My pre
TRT LH=5.7
that is a midle of the range, so my testis and Adrenals are slow.
I am keeping my testis primed as much as possible.
When I am done with other health projects, I plan on increasing my HCG dose just to see what happens.
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When using HCG, at certain point E2 increases to high level.
I would like to understand the process,
to figure if I can expect rise in E2 when I increase HCG dose.
What is produced by testis:
either
testosterone and E1 and E2
or
testosterone and aromatase
then, next pahse, the aromatase convert T to E1 + E2
.
.
If it is the latter, then testis that do not produce T may still produce aromatase (when stimulated)
and cause high E2 without the benefit of making T.