Finasteride is a steroid/T analogue? Could this explain post-Fin HTPA shutdown?

[madclown]

From PAGE 2 of:
http://www.fda.gov/cder/foi/nda/97/2..._BIOPHARMR.PDF

"Finasteride is a synthetic structural analog of Testosterone (T)."


Am I interpreting the above correctly in that Finasteride acts as a steroid/extraneous Testosterone, and so it shouldn't be any surprise when some people's T/LH/FSH crash when they come off it -- just like someone that takes TRT finds their own T production shuts down?

Also, unlike AAS users, Finasteride users don't cycle for 6-8 weeks at a time and take it for months, if not years indefinitely.

Could the above statement be why men experience post-Fin HTPA shutdown? Because in essence we were taking a form of extraneous T/steroids, without cycling and without PCT after coming off?

Would hCG and Clomid not be the solution to correct this (besides lowering E2 if it elevated)?

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On another note, the drug is also listed as an anti-androgen category (PAGE 5): http://www.fda.gov/cder/foi/nda/97/2...1MG_PHARMR.PDF

... despite Merck claiming it is NOT an anti-androgen: Hair loss drug benefits in a 5-year clinical study

WTF? Something doesn't add up here.