High E Post T Use? I don't believe it!

[Ramstein II]

This question comes at you with a bit of disbelief in what is being commonly said. It is often said that after an AAS cycle (PCT) or during TRT recovery E is high or at least out of its normal ratio with T. Now, I can understand the out of ratio with T idea, but I see no reason that E would be "high," that is higher than normal during PCT or Restart. Why would it be?

Ok, there's the argument that aromatized T resulting in E would cause elevated E. Yes, that's true while you take in exogenous T,especially if its supraphysiological. But as the exogenous T is discontinued the E should subside corespondingly. E is made through T aromatization, so if natural T is low post cycle, then so should E.

see this link to an article quoting a study and showing on a graph LH increasing in direct correlation to exogenous T tapering off. Wouldn't this not occur if E was high. I've heard some say that E shuts down FSH and exogenous T shuts off LH, but wouldn't E also shut off LH? I just don't buy the high E argument.

http://www.bodybuilding.com/fun/par31.htm

Also, I've seen in numerous studies involving T use for contraception that FSH and LH rapidly return to normal within 8-12 weeks after T use. This study shows FSH, LH, and T levels backto normal within 8 weeks of recovery after 100mg / wk T cyp use with no SERMS or other PCT. E is not so "out of ratio" or high that it prevented this.

http://jcem.endojournals.org/cgi/reprint/88/3/1167.pdf

I hope someone can explain this. I think this idea comes from bbers who have high levels of aromatized T. I guess I'm just surprised by all the PCT and recovery threads when everything I've read about HPTA suggests that it's pretty resiliant and recovers on its own.