**The Advanced NEVER-ENDING Cycle: ULTIMATE Muscle EXPLOSION!**

[TheMindOfROSS]

Author: THE Ross

Nobody WANTS to COME OFF of steroids.

It SUCKS!

So why DO IT?

WHY STOP STEROIDS?

The ONLY reason to discontinue using steroids is because CERTAIN STEROIDS are inhibitory to the HPTA. Thus, prolonged use of CERTAIN steroids that CAUSE HPTA SHUTDOWN can POSSIBLY lead to secondary HYPOGONADISM--where the testes can no longer manufacture testosterone. Any other DANGERS that steroids alledgedly pose have NOT BEEN SUBSTANTIATED, as was reported in REAL SPORTS, HBO. Ergo, so long as one regularly visits their physician, and blood values remain within normal, there is NO REASON TO COME OFF OF STEROIDS. Furthermore, almost all of the clinical studies conducted on steroids used androgen treatment courses that were MUCH longer than typical steroid cycles advocated on the internet. 6-12-24 month androgen treatment durations.

KEEP 100% OF YOUR GAINS!

Do NOT discontinue anabolic steroids ABRUPTLY!

Post-cycle regimens containing Aromatase Inhibitors and SERM's are simply not enough for the SERIOUS bodybuilder to maintain his muscular gains post-cycle. Once a bodybuilders reaches a certain point of muscular development, the continued use of a mild anabolic becomes justified..


The ONLY THING THAT MATTERS--is that your PITUITARY is NOT INIHIBITED for PROLONGED periods of time. YOUR TESTICLES CAN be inhibited for ANY extended period of time, so LONG AS THE PITUITARY STILL FUNCTIONS!

FACT--To AVOID secondary hypogonadism, we can CYCLE SUPRESSIVE steroids, WHILE REMAINING ON non-inhibitory steroids such as Anavar, Primobolan, Masteron, Turinabol, EQ...SO MANY OPTIONS!

REMEMBER*, NOT ALL STEROIDS CAUSE HPTA SHUTDOWN!

"Shutdown", is defined by a COMPLETE inhibition of the Pituitary/Testes, resulting in a TOTAL cessation of endogenous androgen production.

SOME androgens will only SUPPRESS endogenous androgen production, resulting in a DECREASED testosterone level, but not a complete shutdown. (Tbol, Var, Wistrol, EQ, Dianabol, masteron, proviron, halo, primo)

Very Androgenic/Progestenic/Estrogenic steroids(Tren, Deca, Drol, Test) cause a COMPLETE shutdown of endogenous hormone production.

The distinction between SUPRESSION and SHUTDOWN is utterly important, as steroids that cause LESS supression of endogenous hormones will allow for greater retention of gains upon ending the cycle, and a quicker, easier PCT.

ON TOP OF THAT--you can CONTINUE TO USE THESE *NON-PITUITARY-INHIBITING compounds while AWAITING PITUITARY RECOVERY!
For example:

Let's say that you run a 12 week Testosterone Propionate/Masteron/Winstrol cycle. A LEAN BULK.(one of my favorites!) it looks like this:

Weeks 1-8: Testosterone Propionate, 75mgs ED
Weeks 1-10: Masteron, 100mgs EOD
Weeks 6-12: Winstrol, 75mgs ED

During THIS cycle, your PITUITARY is ONLY inhibited from weeks 1-8, due to the Testosterone Propionate. HOWEVER, from weeks 8-12, your PITUITARY BEGINS TO RECOVER! We want to REMAIN on the MASTERON and WINSTROL during this time, as we NEED this additional anabolic SUPPORT now that we DISCONTINUED TESTOSTERONE.

At week 10 we DISCONTINUE the MASTERON, now ONLY running Winstrol. At this point, the PITUITARY is recovering, and the androgen level in the body has almost normalized(just a tad of Winstrol).

By the end of week 12, YOU WILL NO LONGER BE SHUTDOWN. Your testosterone level will have risen, but not completely recovered. This is NOT IMPORTANT THOUGH, as the ONLY THING THAT MATTERS IS THAT YOUR PITUITARY IS NOT INIHIBITED FOR PROLONGED DURATIONS OF TIME. During your "4 WEEK OFF PERIOD", your Pituitary function was restored, and now, IF YOU SO CHOOSE, you can begin a NEW cycle.

I WOULD SUGGEST NOT going right back on a SUPRESSIVE compound, but rather, continue to utilize a NON-inhibitory compund such as Anavar, Dianabol, or Primobolan for several weeks thereafter. THEN, after about 6-12 weeks of NON-Inhibitory compounds, ONE may choose to start again.

BOTTOM LINE--as long as you COME OFF of SUPRESSIVE compounds, at least every 12 weeks, you can continue to CYCLE STEROIDS YEAR ROUND.
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Horm Metab Res. 1984 Sep;16(9):492-7.Related Articles, Links

Effect of non aromatizable androgens on LHRH and TRH responses in primary testicular failure.

Spitz IM, Margalioth EJ, Yeger Y, Livshin Y, Zylber-Haran E, Shilo S.

We have assessed the gonadotropin, TSH and PRL responses to the non aromatizable androgens, mesterolone and fluoxymestrone, in 27 patients with primary testicular failure. All patients were given a bolus of LHRH (100 micrograms) and TRH (200 micrograms) at zero time. Nine subjects received a further bolus of TRH at 30 mins. The latter were then given mesterolone 150 mg daily for 6 weeks. The remaining subjects received fluoxymesterone 5 mg daily for 4 weeks and 10 mg daily for 2 weeks. On the last day of the androgen administration, the subjects were re-challenged with LHRH and TRH according to the identical protocol. When compared to controls, the patients had normal circulating levels of testosterone, estradiol, PRL and thyroid hormones. However, basal LH, FSH and TSH levels, as well as gonadotropin responses to LHRH and TSH and PRL responses to TRH, were increased. Mesterolone administration produced no changes in steroids, thyroid hormones, gonadotropins nor PRL. There was, however, a reduction in the integrated and incremental TSH secretion after TRH. Fluoxymesterone administration was accompanied by a reduction in thyroid binding globulin (with associated decreases in T3 and increases in T3 resin uptake). The free T4 index was unaltered, which implies that thyroid function was unchanged. In addition, during fluoxymesterone administration, there was a reduction in testosterone, gonadotropins and LH response to LHRH. Basal TSH did not vary, but there was a reduction in the peak and integrated TSH response to TRH. PRL levels were unaltered during fluoxymesterone treatment.(ABSTRACT TRUNCATED AT 250 WORDS


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Talk about stupid ass advice...