The following was cut from another website it was written buy a guy that has given me alot of solid advice
Now to the research supporting using testosterone to taper off with:
Studies of using testosterone for a male contraceptive have shown that it does not cause azoospermia in all men and definitely not at lower dosages (below 100mg
test E per week) (Masumoto, 1990) (Armory, et al., 2001). That is the reason
test E has never been used for a contraceptive. The fact is that even while you are on cycle your body is still active to a certain degree secreting LH. Depending on what steroid(s) you are using i.e. the degree of androgenicity/ how strongly a steroid binds to the AR and the ease in which a compound is aromatised will govern to what degree your hpta is still active (Winters,et al., 1979).
Studies have actually proved this, where using 50mg of
test enanthate on a weekly basis in normal men only lowered FSH and LH secretion by 50% when compared to the placebo group, and larger doses of 100mg and 300mg per week though found to be suppressive, were equally inconsistent in causing azoospermia (Masumoto, 1990) (Armory, et al., 2001). Doses of 25mg of testosterone enanthate had no effect on FSH or LH levels or sperm production compared with placebo (Masumoto, 1990).
A good example of this that many of you have probably found is if you are on cycle using lighter compounds such as
Primo, Anavar, smaller amounts of
Test, Equipoise, Winstrol, Tbol, Dbol, e.t.c. your testicles may not be in a fully atrophied state, however, if you switch your drugs to nandrolone, or trenbolone (which bind strongly to the AR), you will see further shrinkage in the testes. This anecdotal evidence clearly backs the above findings: Some steroids are more suppressive and cause greater shutdown! That of course is why I recommend waiting six weeks at a static 100mg of
test E per week, to clear these steroids and their derivatives from your body before gradually decreasing the testosterone dose.
Further, there is evidence to show that using an anti-E concurrently with 100 mg of
test E per week, so that E is prevented from binding with the receptors in the Hypothalamus prevents any shutdown of sperm production at all (Naftolin, et al., 1973)(Winters, et al., 1979). Highly anabolic agents such as halo, e.t.c at these low doses where shown to do similarly without even the use of an anti E, as these drugs do not aromatize (Winters, et al., 1979).
So, if you want to regain testicular size during a cycle, simply plan in your cycle to remove the highly androgenic compounds from your body (i.e. compounds that aromatise well and bind well to the AR), switching to compounds that have a higher anabolic ratio and concurrently using clomid, and you will ultimately improve the testicular size.
Absolutely no HCG is needed to accomplish this!
So anyways to wrap up, as I said before, the hpta is not fully suppressed when using testosterone in weekly doses below 100 mg of Enathate per week, if used concurrently with an anti E. The goal is to keep estrogen in physiologically normal, or slightly lower than normal levels, or else use clomid or nolva, which antagonize the ER. By doing this you can actually stave off 100% of hpta suppression, while still using 100 mg of
test Enathate a week according to the literature.
If you wait the six week static waiting period, using 100mg of
Test enathate per week while allowing other non testosterone compounds you may have used during your cycle to clear- (which I might add is like a natural taper - allowing your body to slowly come down from being on say greater than 1 gram of
AAS per week) and then you begin to taper your dose from 100 mg per week using an anti E concurrently to ensure the hpta is capable of being active....
Each week as you continue to taper down your dose of
test, the amount of FSH/LH secreted in your pituitary will increase, and the amount of natural
test will increase as well. As LH increases, Sperm production increases, which increases the metabolic demands of the testes, and blood flow then dramatically increases to the area in response to metabolic demands causing hypertrophy - and usually some discomfort - actually some have had to use ice to the area to relieve the discomfort!
Tapering off your anti-E:
As you get down to the 50-25mg per week range you should be tapering off the anti-E as it will no longer be needed to keep your hpta active according to the literature. This is necessary to do on another front, as you need to up-regulate the ER so that it isn't super sensitive to small amounts of estrogen when you finally go off causing late-onset gyno, and even complete shut down... Chronic use of anti-E's causes decreases in estrogen exposure, and just like any drug, if you don't use it for a while, your body becomes more sensitive to it's effects. This means you need to slowly reacclimatize your ER to normal amounts of estrogen aromatisation. You accomplish this by tapering your anti-E so that you should be completely off it by the week you are using only 25mg, which research shows causes absolutely no hpta suppression whatsoever.
So anyways, by the end of the taper you can see now how the testes will have had plenty of time to increase in size. If you didn't use hcg, you can also see how the testes would have been almost hyper-responsive to your own natural LH production when it actually 'kicked into gear'.
And of course the six week waiting period on 100mg of
test E is important, as it doesn't matter what pct method you use, if you still have levels of
AAS in your system and their by-products, it doesn't matter what you do, you can't recover yet until they are cleared.
So basically the taper does the following:
-It gives your body time to adjust from being exposed to a lot of hormones to being just on normal physiological amounts vs the cold turkey approach
-It allows non-testosterone
AAS to clear your body, while you still maintain your size and your libido and workout intensity doesn't have to change
-It allows 'lag-time', for the testes to respond, and your body to adjust back to normal amounts of testosterone.
-At no time does the level of testosterone in your body ever fall bellow physiological norms.
-At no time should you expect to lose your libido
-Best of all you can count the entire taper period as being 'off steroids' as technically you are in the normal physiological range of blood testosterone levels for the entire time you are doing the six week taper. (not the six week waiting period).
Therefore you can return to another cycle in six-week time- and expect good gains.
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References
Armory, J., Anawalt, B., Bremner, W., Matsumoto, A., (2001) Daily Testosterone and Gonadotropin Levels are Simmilar in Azoospermic and Nonazoospermic Normal Men Administered Weekly Testosterone: Implications for Male Contraceptive Development. Journal of Andrology, 22(6). 1053-1060
Matsumoto, A., (1990) Effects of chronic Testosterone Administration in Normal Men: Safety and Efficacy of High Dosage Testosterone and Parallel Dose-Dependant Suppression of Luteinizing Hormone, Follicle Stimulating Hormone, and Sperm Production*. Journal of Clinical Endocrinology and Metabolism, 70(1). 282-287
Naftolin, F., Judd, H., Yen, S., (1973) Pulsatile Patterns of Gonadotropins and Testosterone in Man: The Effects of Clomiphene With and Without Testosterone. Journal of Clincal Endocrinology and Metabolism, (36)1. 285-
Winters, S., Janick, J., Loriaux, L., Sherrins, J., (1979) Studies of Sex Steroids in the Feedback Control of Gonadotropin Concentrations in Men. II. Use of Estrogen Antagonist Clomiphene Citrate*. Journal of Clinical Endocrinology and Metabolism, 48(1). 222-234
My 1st Cycle 
